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Baseline levels of GHK-Cu in the human body peak at roughly 200 ng/mL during late adolescence (around age 20), but suffer a precipitous and permanent decline as an individual ages, dropping to approximately 80 ng/mL by age 60

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Notably, animal models exposed to the fragment exhibit elevated fatty acid oxidation rates without the corresponding hyperinsulinemia or insulin resistance typically induced by intact hGH administration

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